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uBio  Web  Results 91 - 100 of about 3233 Scientific:    Larus atricilla (Laughing Gull)  Synonyms:    Herpetotheres cachinnans (Laughing Falcon)     Streptopelia senegalensis (Laughing Turtle-Dove)  Broader Terms:    Columbiformes (doves)     Laughing     Streptopelia (turtle-doves)  More Specific:    Streptopelia senegalensis aegyptiaca     Streptopelia senegalensis cambayensis     Streptopelia senegalensis ermanni     Streptopelia senegalensis phoenicophila     Streptopelia senegalensis senegalensis     Streptopelia senegalensis sokotrae      Latest Articles on Laughing Gull from uBioRSS Laughing Dove - Aves - Birds Photo Pool Gaivota risonha... - Aves em Portugal Photo Pool Streptopelia senegalensis McMammal - BioLib External Resources: 91.  Reliability of self-reported data on social media versus National Residency Match Program charting outcomes for dermatology applicants. Hu S, Laughter MR, Dellavalle RP Journal of the American Academy of Dermatology, 2020 http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=0 92.  Laughter yoga reduces the cortisol response to acute stress in healthy individuals. Meier M, Wirz L, Dickinson P, Pruessner JC Stress (Amsterdam, Netherlands), 2020 http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=0 93.  Effects of a laughter programme with entrainment music on stress, depression, and health-related quality of life among gynaecological cancer patients. Lee YJ, Kim MA, Park HJ Complementary therapies in clinical practice, 2020 http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=0 94.  Characterizing spine issues: If offers novel therapeutics to Angelman syndrome. Yang X Developmental neurobiology Dev Neurobiol Characterizing spine issues: If offers novel therapeutics to Angelman syndrome. 200-209 10.1002/dneu.22757 Angelman syndrome (AS) is a rare neurodevelopmental disorder characterized by severe mental retardation, microcephaly, speech impairment, frequent epilepsy, EEG abnormalities, ataxic movements, tongue protrusion, bursts of laughter, sleep abruptions, and hyperactivity. AS results from loss of function of the imprinted UBE3A (ubiquitin-protein ligase E3A) gene on chromosome 15q11-q13, including a mutation on the maternal allele of Ube3a, a large deletion of the maternally inherited chromosomal region 15q11-13, paternal uniparental disomy of chromosome 15q11-13, or an imprinting defect. The Ube3a maternal deleted mouse model recaptured the major phenotypes of AS patients include seizure, learning and memory impairments, sleep disturbance, and motor problems. Owing to the activity-dependent structural and functional plasticity, dendritic spines are believed as the basic subcellular compartment for learning and memory and the sites where LTP and LTD are induced. Defects of spine formation and dynamics are common among several neurodevelopmental disorders and neuropsychiatric disorders including AS and reflect the underlying synaptopathology, which drives clinically relevant behavioral deficits. This review will summarize the impaired spine density, morphology, and synaptic plasticity in AS and propose that future explorations on spine dynamics and synaptic plasticity may help develop novel interventions and therapy for neurodevelopmental disorders like AS. © 2020 Wiley Periodicals LLC. Yang Xin X 0000-0003-2153-256X Division of Life Science, Hong Kong University of Science and Technology, Hong Kong, China. eng Journal Article Review 2020 05 25 United States Dev Neurobiol 101300215 1932-8451 IM Angelman syndrome Ube3a iPSC spine synaptic plasticity 2019 09 27 2020 05 01 2020 5 8 6 0 2020 5 8 6 0 2020 5 8 6 0 ppublish 32378784 10.1002/dneu.22757